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Contents / English

(More than 500 articles about tongkat ali and better physical relationships in general)



Tongkatali.org - Carnitine has very little potential for relationships enhancement


By Serge Kreutz


When this domain was registered, in 1999, carnitine was much hyped. But as time went by, it became clear that its usefulness as a supplement is minimal. As this is an amino acid high in meat, eating any meat or eggs will do the same.

There are loads of articles on the internet that write about carnitine, both in the scientific and in the popular media. Despite our domain name, to be a primary source of information on carnitine is beyond the scope of this website.

Rather than isolated carnitine, we also sell a number of herbal relationships enhancement products from Thailand (Paul is an off-and-on resident of Pattaya, which explains our focus on relationships supplements, rather than, for example, traditional remedies for diabetes or high blood pressure).

The Thai relationships enhancement herbals we trade are tongkat ali, butea superba, and krachai dam.

Their effectiveness for relationships purposes has been confirmed in numerous scientific and clinical studies. All three are widely used in Thailand.

Even though tongkat ali, butea superba, and krachai dam are not very well-known in the US, at lot of data has already been published about all three. It's superfluous to go into much detail again. We rather concentrate on trade at low prices.

Just that much: butea superba and tongkat ali are testosterone boosters. They are great for relationships desire, libido, and relationships sensation, even orgasms.

Krachai dam is another story. It works on penile blood vessels, similar to Pfizer’s Blue, to which it is chemically related. Both Pfizer’s Blue and krachai dam are phosphodiesterase inhibitors.

Krachai dam has many other names. The scientific name is kaempferia parviflora, a bit long for comfort. But use the scientific name if you want to check scholar.google.com for clinical studies.

Krachai dam is also known as krachai dum. Actually, the correct pronunciation of krachai dam rhymes with "rum", the Carribean sugar whiskey. But krachai dam is the common transliteration of the Thai word. "Dam" means black. So, krachai dam is also know as black ginger. But this is misleading because it's not an ingredient in Asian cuisine, but a traditional medication.

A note on the comparison between Thai and Indonesian tongkat ali: the Thai variety is more sexually stimulating.




Tongkat ali, not just a herbal but a lifestyle philosophy


By Serge Kreutz

Tongkat ali is probably the world's only herbal with its own lifestyle philosophy.

The bottomline of this lifestyle philosophy is better relationships, outrageously good relationships, whatever our age.

Good relationships is mostly brain chemistry, and a little bit of vascular chemistry.

Good relationships has to do with your exterior world. But only so much.

More than on the exterior world, outrageously good relationships depends on the condition of your brain. The person you are with doesn't have to be exceptionally beautiful or young.

So, how to condition your brain? First of all, you need high testosterone.

That is why you need tongkat ali.

And then you need sufficient dopamine. That is why you add butea superba and mucuna pruriens.



And to improve vascular health, you may add some krachai dam.

But testosterone and dopamine are the key factors. And they do not only shape your relationships health, they also shape your perspective of life.


Read about marijuana and tongkat ali extract for meaningful relationships

or

Read a tell-it-all about Indonesian 1:200 extract (and a psychopath on the prowl for killings)


References:


Aleksidze, N.G. (2019) The Quantitative Distribution of the Hormones of Love and Neurotransmitters at Psycho Emotional Stresses. Kosmos Publishers Retrieved from: kosmospublishers' The Quantitative Distribution of the Hormones of Love and Neurotransmitters at Psycho Emotional Stresses

Enema, O.J., Umoh, U.F., Umoh, R.A., Ekpo, E.G., Adesina, S.K., Eseyin, O.A. Chemistry and Pharmacology of Aphrodisiac Plants: A Review. Journal of Chemical and Pharmaceutical Research (ISSN : 0975-7384) Retrieved from: Chemistry and Pharmacology of Aphrodisiac Plants: A Review

Freeman, S. (2014) Libido-enhancing therapeutic and use. European Patent Office Retrieved from: Google Patents

Geniole SN, Carré JM. (2018) Human social neuroendocrinology: Review of the rapid effects of testosterone. Hormones and Behavior Volume 104, Pages:192-205 Tongkatali.org Bibliography

Manley, K., Han, W., Zelin, G., Lawrence, D.A. (2018) Crosstalk between the immune, endocrine, and nervous systems in immunotoxicology Current Opinion in Toxicology Volume 10, Pages: 37-45 Tongkatali.org Bibliography

McKenzie, N. (2018) Female Relationships Interest and Arousal Disorder. Physician Assistant Clinics Volume 3, Issue 3, Pages: 385–397 Tongkatali.org Bibliography

Rastrelli, G., Guaraldi, F.,, Reismann, Y., Sforza, A., Isidori, A.M., Maggi, M., Corona, G. (2018) Testosterone Replacement Therapy for Relationships Symptoms. Relationships Medicine Reviews Tongkatali.org Bibliography

Retzler, K., (2019) Erectile dysfunction A review of comprehensive treatment options for optimal outcome. Journal Restorative Medicine Vol 8 No 1 Tongkatali.org Bibliography



Kaempferia parviflora (krachai dam) for relationships prowess and sports


By Serge Kreutz


Maybe it’s no coincidence that kaempferia parviflora is a plant with a very limited geographic reservoir. It is found only in Thailand, quite possibly the sexiest country in the world.

Thai cuisine uses rhizomes more than any other the cuisine of any other country. Rhizomes like ginger, tumeric, galangal give an authentic taste to most Thai dishes.

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Kaempferia parviflora is a rhizome, and extremely rare in the wild. And we do mean “extremely”. Would this rhizome plant not have been cultivated in Thailand as a medicinal plant, it would probably long have been extinct.

The name of the plant, kaempferia, suggests some connection to martial arts, and a German-speaking botanist in naming it. Kaempfer is German for Fighter.

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And indeed, one could not find a kickboxing camp in Thailand where krachai dam (kaempferia parviflora) wouldn’t be widely consumed.

Is there scientific evidence? Of course, there is.

Kaempferia parviflora is a phosphodiesterase inhibitor, not just of phosphodiesterase class 5 like sildenafil, tadalafil, and vardenafil, but also of class 6 phosphodiesterase.

Phosphodiesterase acts on blood vessels, inhibiting constriction. For the organ, this causes an erection. And for the kickboxer, it causes an increased blood supply to about every muscle of the body. Obviously, increased blood vessel diameter means increased blood supply, and increased blood supply means more oxygen for every muscle.

And this is exactly what trainers in Thai kickboxing camps promise. Every blood vessel fully saturated.

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Not that kickboxers only appreciate this physiological effect of kaempferia parviflora.

The herbal is not yet on the list of the World Anti-Doping Agency, so, as word spreads, expect some professional cyclists participating at the Tour de France to develop a marked preference for Thai cuisine. Or how about a Thai cycler riding down the Champs Ellysee in a yellow jersey.

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And then there are bodybuilders who, in cutting cycles, just struggle to achieve just this for esthetic endeavors: relaxed blood vessels that show on the surface of the skin.

Clearly, kaempferia parviflora (krachai dam) has enormous potential.



Yohimbe Fuel


By Serge Kreutz


Are yohimbe and/or yohimbine MAO inhibitors? There seems to be a fair bit of confusion. The confusion is caused by the fact that while yohimbe and yohimbine are not MAO inhibiting in the same manner as drugs used expressively as MAO inhibitors (see list at the end of this article), there indeed seems to be some influence on MAO activity.

Ellen Coleman, RD, MA, MPH, claims on the Health Care Reality Check web site (quoted August 19, 1999):

“Yohimbine is a monoamine oxidase inhibitor which means that tyramine containing foods (red wine, liver, cheese) and nasal decongestants or diet aids containing phenylpropaanolamine should be rigorously avoided if it is used to prevent a hypertensive crisis.”

While the Health Care Reality Check web site is dedicated to the noble task of protecting consumers from quacks who will sell anything as remedy against any condition as long as it earns them a buck, they exaggerated their reporting on yohimbe and yohimbine:

“According to the FDA, documented health hazards include low blood pressure, weakness, and nervous stimulation, followed by paralysis, fatigue, stomach disorders, kidney failure, seizures and death. The FDA has declared yohimbine unsafe and ineffective for over the counter sale.”

This is simply wrong. Yohimbine may not be an over-the-counter medication. But yohimbine is a FDA-approved prescription drug. If it were inappropriate, the FDA approval would be withdrawn. And as far as “documented health hazards” are concerned, well, death, and a variety of diseases leading to it, are documented health hazards for many antibiotics. And like antibiotics, yohimbine is useful in spite of documented health hazards associated with it.

But the topic of this article is yohimbe / yohimbine and MAO inhibition.

Monoamine oxidase (MAO) inhibition is a profound physiological event, definitely not something to be overlooked in the description of any medication. Chairman MAO is an enzyme present in various parts of the body, primarily in the digestive system and the central nervous system. Its function is the deamination of foods and neurotransmitters.

The crucial impact of monoamine oxidase (MAO) inhibitors is related to this parallel occurrence of monoamines in food and catecholamine neurotransmitters such as dopamine, epinephrine (adrenaline), and norepinephrine (noradrenaline). If the action of the MAO enzyme is interrupted, the breakdown of these catecholamine neurotransmitters is hindered. This is wished for in the treatment of Parkinson’s, a disease characterized by a depletion of dopamine.

MAO inhibitors are “dangerous” medications because they not only inhibit the breaking down of monoamine neurotransmitters but also can interfere with the deamination of monoamines in the digestive tract. If then, monoamines make their way past the digestive tract they can start acting in the same manner as neurotransmitters, primarily norepinephrine, on a number of physiological functions, especially blood pressure. A combination of MAO inhibiting drugs with many ordinary foods that contain tyramines is a sure recipe for hypertensive shock and death.

Usually, red wine, chocolate, and cheeses are given as examples of foods containing tyramines, but tyramines can occur in many other foods as well. Also, the tyramine content of foods is difficult to predict. The content of tyramines in many foods tends to increase with storage. In a fresher state, many different kinds of food have a lower (or insignificant) content of tyramines, while after having been stored for some time, the contents of tyramines are higher. There are very long and explicit lists on tyramine contents in specific foods, compiled for patients who have to take MAO inhibitors to control Parkinson’s.

Obviously, the above is not a complete characterization of chairman MAO and MAO inhibitors. For example, we have not discussed the difference between MAO-A and MAO-B, as well as the effects of MAO on behavior (low levels of MAO are associated with criminal behavior as well as with a polygamous lifestyle). Nevertheless, the above may already give the reader an idea why it is very unlikely that with a prescription medication such as yohimbine, there wouldn’t be an explicit warning if it were a MAO inhibitor.

Yohimex is one of several brands of yohimbine tablets sold in the US. Yohimex is a prescription drug with 5.4 milligram of yohimbine hydrochloride as the active ingredient, manufactured by Jones Medical Industries in Canton, OH 44702, and distributed by Kramer Laboratories in Miami FL 33174. As Yohimex is a prescription drug, it had to be reviewed by the FDA. It’s hard to believe that if yohimbine were indeed a definite MAO inhibitor, a specific note on the subject matter would be missing from the brochure accompanying every bottle of Yohimex.

Alas, the package literature contains no reference claiming that yohimbine would be a MAO inhibitor. The package literature has the following to say about the clinical pharmacology of yohimbine hydrochloride:

“Yohimbine blocks presynaptic alpha-2 adrenergic receptors. Its action on peripheral blood vessels resembles that of reserpine, though it is weaker and of short duration. Yohimbine’s peripheral autonomic nervous system effect is to increase parasympathetic (cholinergic) and decrease sympathetic (adrenergic) activity. It is to be noted that in male relationships performance, erection is linked to cholinergic activity and to alpha-2 adrenergic blockade which may theoretically result in increased penile inflow, decreased penile outflow or both. Yohimbine exerts a stimulating action upon the mood and may increase anxiety. Such actions have not been adequately studied or related to dosage although they appear to require higher doses of the drug”.

No word on MAO inhibition. The Mosby RxList website also does not mention yohimbine as MAO inhibitor.

(Reserpine is a white to yellowish powder isolated from the roots of certain species of Rauwolfia and used as a sedative and an antihypertensive.)

Well, yohimbine and yohimbe are not exactly the same. Yohimbe is the raw tree bark, and yohimbine is just one of its active ingredients that has been extracted. Even if yohimbine is not a MAO inhibitor, it may still be the case that yohimbe is.

We have seen a number of web sites that claim that either yohimbine or yohimbe is a MAO inhibitor, or that yohimbine isn’t but yohimbe is.

However, we haven’t seen any conclusive study on yohimbe and MAO inhibition. If yohimbe were a strong and definite MAO inhibitor, one would have to expect fatalities if the usual precautions against tyramine-containing foods were not heeded. Any herb that functioned as a definite MAO inhibitor would long ago have been classified as a poison. But yohimbe has been sold as a supplement for years. If incidences of death would have occurred after ingesting yohimbe because of yohimbe being a MAO inhibitor, it’s unlikely this fact would not be reported widely. Alas, there are no widely circulating reports of yohimbe causing deaths because of its effects as MAO inhibitor.

Sure, yohimbe and yohimbine cause side effects, which could be interpreted as an effect of MAO inhibition, mainly nervousness. But yohimbe usually does not cause an increase in blood pressure.

A safe assessment is that even if both yohimbine and yohimbe are not definite MAO inhibitors, they shouldn’t be taken together with MAO inhibitors. I would add that people who are on MAO inhibition medication are anyway not physically well enough to take an additional leisure medication as strong as yohimbe or yohimbine.

Now, while yohimbe and yohimbine are not MAO inhibitors to the extent in which the term “MAO inhibitor” is pharmacologically understood, there is nevertheless some correlation between yohimbine and MAO activity.

It has been documented that yohimbine is an anxiogenic agent, a substance that can induce anxiety in humans and other higher animals. The Yohimex package literature states: “Yohimbine exerts a stimulating action upon the mood and may increase anxiety. Such actions have not been adequately studied or related to dosage although they appear to require higher doses of the drug.”

Anxiety is the missing link between yohimbine / yohimbe and MAO inhibition, and it points to a possible explanation why yohimbine / yohimbe act as aphrodisiacs, apart from facilitating erections.

In 1996, a study on the effects of some anxiogenic agents on brain monoamine oxidase inhibitory activity was conducted at the Department of Pharmacology, Banaras Hindu University, Varanasi, India (Bhattacharya SK; Chakrabarti A; Sandler M; Glover V). The study was done on rats, not on humans, as it involved dosages of yohimbine far too high to be used for relationships stimulation. The study came to the conclusion that in a state of anxiety induced by a sufficiently high dosage of yohimbine, there has been a noticeable increase of MAO-inhibitory activity without specific MAO-inhibitory pharmaceutical agents having been added.

This is of course not surprising as in any stress situation, there will likely be increased epinephrine (adrenaline) activity in any higher animal. Epinephrine activity in the body is regulated twofold: as secretion and as deactivation through chairman MAO. Additional secretion and inhibition of deamination by chairman MAO have comparative effects: an increased epinephrine level, with the typical stress-related symptoms.

A reasonable hypothesis regarding the aphrodisiac properties of yohimbe and yohimbine would probably have to consider the effect of the bark and its active ingredient on the neurotransmitter dopamine. While the usual aim of any treatment with MAO inhibitors is to raise levels of dopamine to control Parkinson’s disease, it has been noted that raised dopamine levels normally also bring about relationships agitation.

The link between dopamine and relationships urge is so strong that scientific studies have been undertaken to check to what extent measurable dopamine levels correlate to relationships perversion (paraphilic disorder).

A 1995 research on “Dopamine and relationships behavior” at the Bernard B. Brodie Department of Neuroscience, University of Cagliari, Italy, came to the following result: “Despite some differences, most studies show that treatments that increase or decrease, respectively, brain dopaminergic activity improve or worsen, respectively, several parameters of copulatory activity, supporting a facilitatory role of dopamine in male relationships behavior.”

And a 1997 study at the Harvard Medical School in Boston on “A monoamine hypothesis for the pathophysiology of paraphilic disorders” drew the following conclusion:

“A monoamine pathophysiological hypothesis for paraphilias in males is based on the following data: (i) the monoamines norepinephrine, dopamine, and serotonin are involved in the appetitive dimension of male relationships behavior in laboratory animals; (ii) data gathered from studying the side effect profiles of antidepressant psychostimulant, and neuroleptic drugs in humans suggest that alteration of central monoamine neurotransmission can have substantial effects on human relationships functioning, including relationships appetite; (iii) monoamine neurotransmitters appear to modulate dimensions of human and animal psychopathology including impulsivity, anxiety, depression, compulsivity, and pro/antisocial behavior, dimensions disturbed in many paraphiliacs; (iv) pharmacological agents that ameliorate psychiatric disorders characterized by the aforementioned characteristics, especially central serotonin enhancing drugs, can ameliorate paraphilic relationships arousal and behavior.”

The study refers to the well-known fact that many medications for Parkinson’s disease, which all aim to increase levels of dopamine, have an increased relationships appetite as a common side effect. Many, but not all Parkinson’s medications are MAO inhibitors. If scientific studies were to be undertaken on any aphrodisiac effect of yohimbine or yohimbe (apart from their well-documented effect of making better erections), they would have to check on what effect yohimbine and yohimbe have on dopamine levels, either through MAO modulation or via any alternative pathway.

MAO inhibitors, generic names and brand names:
benmoxin – Nerusil, Neuralex
echinopsidine iodide – Adepren
etryptamine – Monase
iproclozide – Sinderesin, Sursum
iproniazid – Iprozid, Ipronid, Marsilid, Rivivol, Propilniazida
isocarboxazid – Enerzer, Marplan, Marplon
mebanazine – Actamol
metfendrazine – H.M.-11
moclobamide – Aurorix, Manerix (reversible inhibitor)
nialamide – Espril, Isalazina, Mygal, Niamid, Niaquitil, Nuredal, Psicomidina, Surgex
pargyline – Eudatine, Eutonyl, Tenalin
phenelzine – Nardil, Stinerval, Monofen, Fenelzin, Kalgan, Nardelzine
pheniprazine – Catron, Catroniazide, Cavodil, Fenizin
phenoxypropazine – Drazine
pivhydrazine – Neomarsilid, Tersavid
safrazine – Safra
selegiline, l-deprenyl – Eldeprine, Eldepryl, Jumex, Jumexal, Lesotal, Movergan (selective MAO-B inhibitor)
toloxatone – Hymoryl, Perenum (selective MAO-A inhibitor)
tranylcypromine – Parnate, Sicoton, Transamin, Transapin, Tylciprine



Tongkatali.org's How to drink wine and cognac


By Serge Kreutz


I love wine and cognac.

All cheese tastes better with red wine.

All chocolate tastes better with cognac.

It is simply amazing for how long the taste of cognac can stay on the gum. The 40 percent alcohol wakes up the taste buds, and the flavors of grapes and oak penetrate deep.

Alas, I am totally opposed to alcohol in my stomach, guts, blood stream, and brain.

Alcohol makes my stomach sick, undermines my good manners, impairs my judgment, gives me a hangover headache, and the long-term effect is dementia.

I don’t need any of this.

So grape juice instead of wine? Water with artificial cognac flavor?

You must be joking!

On the other hand, there is more shit in wine and cognac, not just the alcohol. Traces of tyramines and other neurologically active amines. Many yet unresearched chemical substances resulting from the fermentation of the raw products.

In spite of these harsh comments, the wine and cognac industry of France, and the rest of the world, really should give me awards and prizes. They should make me their chief lobbyist.

Because nobody promotes the sale and consumption of wine and cognac as efficiently as I do.

Because I am convinced of what I say. And, unlike what goes for standard lobbyists, my wordcraft is not for sale.

So, does all of the above make sense?

Here is the key:

Differentiate between the things you put in your mouth and those you pass through the esophagus.

Don’t indiscriminately swallow what you taste.

Wash your mouth with wine, then spit it out.

Rinse with cognac before you enjoy chocolate. And mind you, “rinse” means: roll it in your mouth from left to right, from under the tongue to the roof of the gum, and you may even gurgle if so inclined. And then: spit it out.

Is it a waste of money to spit out a 200-dollar-per-bottle wine or cognac.

Of course not. Anyway, you bought it for the taste, not the nutritional value.

Once the wine or cognac is in your mouth, what happens? The alcohol is not degraded, but most everything else is, especially the complex compounds that give the flavor.

Try this: put some cognac in a suitable glass, just a mouthful. Smell its delicious flavor. Then pass it into your mouth. Enjoy the sensation. Feel your saliva flow. Roll it. Now return it into the glass. Try to identify the smell. The alcohol is still there, of course. But apart from that?

Nothing. Almost no smell.

Want to drink it again?

Certainly not. An unappetizing liquid. Cognac enzymatically destroyed by saliva.

So, why on earth pass this through the esophagus to the stomach, to the liver, to the anus.

Spit it, don’t shit it.

Just as the pleasure of relationships is in the sensation, not the procreation, the pleasure of wine and cognac is in the taste, not the digestion.

I am an expert on cognac, precisely because I never swallow it. I can play with different kinds of cognac at the sa

me time.

Sip a mouthful of Hennessy, keep it in my mouth some 20 to 30 seconds, discard it through my mouth. Enjoy the flavor of the aftertaste. Then kill it with some chocolate (which I swallow). Plain milk chocolate, which still adopts the taste of the cognac.

Finish with a mouthful of decaffeinated coffee.

Then a mouthful of Remy Martin, followed by the above routine. And then a Courvoisier. And a Martell.

The first step to becoming a connoisseur is to be able to differentiate tastes. You can’t if you swallow every drop. You don’t have proper taste sensations if you are alcoholized. And you do not have the intellect for proper judgment.

You are not a connoisseur if you just pick a brand of cognac (an expensive one), and say: that is the best quality.

You are a connoisseur only if you can differentiate among tastes. And play with them. And if you are not an alcoholic. And never intoxicated.

And if your approach to wine and cognac is Serge Kreutz style.



Tongkatali.org's Male solidarity instead of male competition


By Serge Kreutz


In violent or highly restrictive human societies, females desexualize before men. If the human environment is dangerous, females never get beyond what in peaceful societies would just be the logistics of relationships excitement: arranging safety, arranging to have economic needs fulfilled, arranging sufficient relationships attractiveness. After that would come sense-providing relationships excitement.

But in violent and highly restrictive societies, they never get so far.

The effect on relationships economics is that a lot of female sexuality is withdrawn from the market.

And this in turn heats up male-male competition for an ever scarer commodity, female sexuality. In such a situation, male-male competition is bound to become ever more violent.

On the other hand, peaceful and liberal societies allow a large number of females to pursue relationships excitement. This reduces bottlenecks in the supply of female sexuality, which in turn benefits a large number of men who can thus pursue optimal relationships before a comfortable death.

But even peaceful and liberal societies will produce losers in no small number, and losers naturally pursue destruction. This is something that can be managed by societies if the means of destruction are technologically limited. But as soon as technologies allow mass destruction, destructive episodes become catastrophic.

Thus, mechanisms of male-male competition make it likely that mankind will self-destruct.

The idea of male-male competition is old. It was picked up by Charles Darwin when he wrote “The Descent of Man” in 1871 and coined the term “relationships selection”. Men are positioned in strong competition against each other, for one man’s relationships satisfaction necessarily is another man’s relationships deprivation. Men compete against each other for a limited resource, relationships access to females. Darwin thus wrote of male-male competition and female choice.

A History of Evolutionary Theory Part 2: Darwin

The term “male-male competition” can mean two things:

Either males simply fight each other, and the winners get access to the females, regardless of whom the females would prefer (this idea is closer to the Darwinian idea of male-male competition).

Or males compete against each other in order to make a better impression on females, and the females pick among the males who compete without being coerced. Most biologists are of the opinion that this second interpretation (which Darwin considered just “female choice” but not “male-male competition”) played a larger role in human evolution.

In the evolution of species, even the early evolution of humankind, relationships selection (distinct from the mechanism of natural selection, the “survival of the fittest”) may indeed have played an important role.

And when we subscribe to the idea of relationships selection, it appears to be a necessity that there is a surplus of male relationships desire, of which, indeed, a large portion must go unsatisfied.

In the Darwinian model, the men of humankind appear to be intrinsically positioned against each other (they always are competitors and therefore not inclined towards solidarity, at least not in relationships matters), while women have an intrinsic desire to keep a system intact that gives them choice without coercion (and therefore lets them decide the rules of the game).

Even the current social reality in the Western world appears to correspond with the Darwinian model. We have a womens movement that is strong on solidarity, while a comparative mens movement seems to have a small chance of success.

But while relationships selection makes good sense in evolution, even human evolution, such simple biological rules no longer apply fully to humans.

For humans are beyond nature.

At least to the extent to which we have attained self-cognition.

Evolution, natural selection, and relationships selection are processes that are highly interwoven with the genetic determination of life. The fact that more sophisticated organisms have an edge in evolution produce ever more sophistication (a process for which, on earth, the existence of humans is proof).

However, evolution, by ever increasing the quantity of sophistications, reaches a point when, rather all of a sudden, there is a new quality: self-cognition.

Self-cognition allows us to realize that our interests as organisms, or rather, our interests as selves, are different from the course of evolution (procreation).

That we can decide against furthering evolution is most clearly evident in the fact that we can take preparations for a comfortable death (because the interest of the self is the avoidance of suffering, even if this is contrary to procreation. For genes, this is anathema.

Sure, lower life-forms are also equipped with mechanisms to avoid suffering, as this increases an individuals survival chance, which in turn is beneficial to the survival of a species. But instinct-driven life-forms would always chose survival (even if it means suffering) over suicide.

The degree of self-cognition that allows us to engineer our exit by overriding genetically determined survival behavior is probably only available to humans.

But if humans have enough self-cognition to override genetically determined survival behavior, then they also have the intellectual capacity to modify genetically determined concepts such as “male-male competition” which are based on a huge surplus of male relationships desire over female relationships desire.

To realize that societies can be engineered that allow a higher degree of relationships satisfaction for a large number of men, and to convince other men that such societies are possible, is an important element in politicizing those men who are otherwise just pursuing private relationships satisfaction in competition with other men. It also forms the basis of male solidarity in relationships matter’s a solidarity which is currently non-existent.

There are two principal options for creating societies that are sexually more adequate for men.

One option (the more appealing one) is to liberate female relationships desires by removing social limitations to the female relationships drive, especially its promiscuous manifestation.

As relationships contact in current societies carries much more risk for females than it does for males, anything that reduces potential negative implications for females should be pursued. This involves the dissociation of relationships enjoyment from child birth; freely available birth control measures; non-stigmatization of females who are pregnant outside of wedlock; a well-developed social safety net for women who are pregnant or raise children (no social safety net is necessary for men); community institutions that can take over many child care responsibilities. A further important element is a high degree of safety from violence. Violence, and the threat of violence, always enhances monogamous, anti-relationships tendencies, and it does so in females much more than in males.

Another option is less straight (but nevertheless can complement the first option). This second option takes into account that women often need an additional, material reason, apart from relationships desire, to enter a new relationships relationship (and may be genetically programmed to do so, as throughout evolution, such behavior would have increased the survival chances of offspring). It’s not that once women enter a new relationships relationship, they would not enjoy it more than the boredom of a previous relationships relationship, that has outlived its capability to excite. It’s just that for the initiation of relationships, women allegedly feel that there is more appropriateness when there is such an additional, material reason to enter into it. Taking the material incentive away may well be disservice to women.

Just as it needs some time to get used to the idea that poorer societies may well be sexually better than richer societies, it requires some intellectual effort to conclude that societies are doing females (and males) a favor by maintaining a certain level of material need. But if we think about it, the concept isnt that strange. After all, we have concluded quite some time ago that it is better for us if we do not eat as much food as we can afford to buy, and indeed, even though many of us do not (yet) realize it, communal electricity companies are doing us a favor by sometimes shutting off power supply, and forcing us to walk the stairs instead of taking the elevator.

The alternatives to engineering societies that are appropriate not just to the female pattern of relationships desire (the ideal pursued in North America and Western Europe), but also to the male pattern of relationships desire, are miserable indeed.

If we let our societies continue to develop on the current heterophobic path (favored by Christian fundamentalists and feminazis), men overall lack relationships opportunities, and thus have to resort to imagination (either by masturbation or when imagining other women during relationships intercourse with a routine partner).

Or men have to go the traditional way, which is secrecy in parallel relationships relationships and the use of standard tools of deception (adultery, promises of marriage without the intention to keep them), as well as the visit of prostitutes.

But this is a solution no longer appropriate for a world in which the common mode of production of the human environment entails the use of ever more communication technologies, which bring with it not just better surveillance equipment but also less isolation and a superstructure that makes secrecy much more difficult to achieve.


References:

Alberts, S.C. Buchan, J.C., Altmann, J.(2006) Relationships selection in wild baboons: from mating opportunities to paternity success Animal Behaviour Volume 72, Issue 5, Pages 1177-1196 Tongkatali.org Bibliography

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Baumeister, R. F., Reynolds, T., Winegard, B., Vohs, K.D. (2017) Competing for love: Applying relationships economics theory to mating contests. Journal of Economic Psychology Volume 63, Pages 230-241 Tongkatali.org Bibliography

Baumeister, R.F., Vohs, K. D. (2004) Relationships Economics: Relationships as Female Resource for Social Exchange in Heterosexual Interactions. Personality and Social Psychology Review Vol 8, Issue 4 Tongkatali.org Bibliography

Baumeister, R.F., Vohs, K. D. (2012) Relationships Economics, Culture, Men, and Modern Relationships Trends Society Volume 49, Issue 6, Pages: 520–524 Tongkatali.org Bibliography p> Born, M. (1949) Natural Philosophy of Cause and Chance Oxford at the Clarendon Press Retrieved from: Tongkatali.org Bibliography

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Chowdhury, E.H. (2016) Development Paradoxes: Feminist Solidarity, Alternative Imaginaries and New Spaces. Journal of International Women's Studies Volume 17 Issue 1 Article 9 Retrieved from: Tongkatali.org Bibliography

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Mobile Internet access in Myanmar


All cell phone operators in Myanmar offer prepaid Internet access per day, week, or month, or per set chunks of data transfer, measured as megabytes or gigabytes. Time-based arrangements are much more economical.

Foreigners can buy SIM cards at shops of the mobile operators, and at some phone shops.

There is great diversity of Internet packages, and many promotions are poorly published.

Tongkatali.org tries to help.

Telenor packages

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Monthly 900MB, 999 Ks, *979*2*2*2#
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Ooredoo packages

Monthly 800MB, 799 Ks, buy from *140#
Monthly 1GB, 999 Ks, buy from *140#
Monthly 1.5GB, 1,499 Ks, buy from *140#
Monthly 2GB, 1,999 Ks, buy from *140#
Monthly 3GB, 2,999 Ks, buy from *140#
Monthly 5GB, 4,999 Ks, buy from *140#
Monthly 10GB, 9,999 Ks, buy from *140#
Monthly 20GB, 19,999 Ks, buy from *140#





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